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Robert V. Tauxe
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Robert V. Tauxe : ウィキペディア英語版
Robert V. Tauxe

Robert V. Tauxe, MD, MPH, USPHS (Ret.) is the Deputy Director of the Division of Foodborne, Waterborne and Environmental Diseases〔(About | CDC DFBMD )〕 of the Centers for Disease Control and Prevention. He received a BA in cultural anthropology and a Masters in Public Health from Yale, an MD degree from Vanderbilt, and is board certified in Internal Medicine. He spent two years as an Epidemic Intelligence Service officer at the Centers for Disease Control and Prevention, before joining the scientific staff at the CDC in 1985. His research interests include bacterial enteric diseases, epidemiology and pathogenesis of infectious diseases, epidemiological and clinical consequences of bacterial genetic exchange, antimicrobial use and resistance to antimicrobial agents, and teaching epidemiological methods. He has served internationally in Belgium, Mali, Rwanda, Peru and Guatemala and has participated in or supervised numerous domestic and overseas epidemiological investigations, including the (Rajneeshee bioterror attack ), the (United States salmonellosis outbreak ), the (Haiti cholera outbreak ), and Pulsenet. His faculty appointments include the Emory University School of Public Health in the Department of International Health, and the Emory University Department of Biology. He has authored or co-authored more than 250 scientific journal articles, letters and book chapters.
In comments on the June 2011 E. coli O104:H4 outbreak in Europe, because three cases had appeared in the U.S., he stated that the supposedly new aggressive enterohemorrhagic and enteroaggregative strain had been seen before in the early 1990s (contrary to some other professional statements of other prominent microbiological experts and physicians in Europe). Although it can produce bloody diarrhea, severe dehydration requiring fluid replacement therapy, and (uremic syndrome )- HUS- which can lead to permanent kidney impairment or failure or death, it is not the same strain and is much newer and likely even more aggressive than the much better known and more common E. coli O157:H7 strain. E. coli strains, both pathological and naturally occurring non-pathological ones, in the human intestinal tract trade genetic characteristics among themselves- and among other microbially related species- fairly easily, allowing for more aggressive and thus more resistant strains to develop. Even if the strain dates back to the early 1990s, it would still be relatively new as a major health concern, and treatment- which is harder for any new and severe strain- is (like for other pathological E. coli strains) mainly supportive.
== References ==




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